Publications
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Filters: Author is Gosselin-Badaroudine, Pascal [Clear All Filters]
, “A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy.”, Sci Rep, vol. 8, no. 1, p. 13804, 2018.
, “Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide.”, FASEB J, vol. 31, no. 7, pp. 3066-3071, 2017.
, “Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4.”, J Gen Physiol, vol. 148, no. 2, pp. 133-45, 2016.
, “Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides.”, Sci Rep, vol. 5, p. 12475, 2015.
, “Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy.”, Channels (Austin), vol. 9, no. 3, pp. 139-44, 2015.
, “Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy.”, J Gen Physiol, vol. 145, no. 2, pp. 93-106, 2015.
, “Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents.”, Front Pharmacol, vol. 6, p. 301, 2015.
, “Biophysics, pathophysiology, and pharmacology of ion channel gating pores.”, Front Pharmacol, vol. 5, p. 53, 2014.
, “Molecular biology and biophysical properties of ion channel gating pores.”, Q Rev Biophys, vol. 47, no. 4, pp. 364-88, 2014.
, “Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?”, Channels (Austin), vol. 8, no. 1, pp. 90-4, 2014.
, “Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutation.”, Front Pharmacol, vol. 4, p. 126, 2013.
, “Gating pore currents and the resting state of Nav1.4 voltage sensor domains.”, Proc Natl Acad Sci U S A, vol. 109, no. 47, pp. 19250-5, 2012.
, “A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype.”, PLoS One, vol. 7, no. 5, p. e38331, 2012.
