Publications
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Filters: Author is Sejnowski, Terrence J [Clear All Filters]
Age dependency of trauma-induced neocortical epileptogenesis. Front Cell Neurosci. 2013;7:154.
Biophysical basis for three distinct dynamical mechanisms of action potential initiation. PLoS Comput Biol. 2008;4(10):e1000198.
Cellular and network mechanisms of electrographic seizures. Drug Discov Today Dis Models. 2008;5(1):45-57.
Pyramidal neurons switch from integrators in vitro to resonators under in vivo-like conditions. J Neurophysiol. 2008;100(6):3030-42.
Nonlinear interaction between shunting and adaptation controls a switch between integration and coincidence detection in pyramidal neurons. J Neurosci. 2006;26(36):9084-97.
Reduction of anion reversal potential subverts the inhibitory control of firing rate in spinal lamina I neurons: towards a biophysical basis for neuropathic pain. Mol Pain. 2006;2:32.
Slow state transitions of sustained neural oscillations by activity-dependent modulation of intrinsic excitability. J Neurosci. 2006;26(23):6153-62.
Homeostatic synaptic plasticity can explain post-traumatic epileptogenesis in chronically isolated neocortex. Cereb Cortex. 2005;15(6):834-45.
Maintenance and termination of neocortical oscillations by dynamic modulation of intrinsic and synaptic excitability. Thalamus Relat Syst. 2005;3(2):147-156.
Membrane bistability in thalamic reticular neurons during spindle oscillations. J Neurophysiol. 2005;93(1):294-304.
Frequency-selective augmenting responses by short-term synaptic depression in cat neocortex. J Physiol (Lond). 2002;542(Pt 2):599-617.
Model of thalamocortical slow-wave sleep oscillations and transitions to activated States. J Neurosci. 2002;22(19):8691-704.
Short- and medium-term plasticity associated with augmenting responses in cortical slabs and spindles in intact cortex of cats in vivo. J Physiol (Lond). 2002;542(Pt 2):583-98.
