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“Regulation of central Na+ detection requires the cooperative action of the NaX channel and α1 Isoform of Na+/K+-ATPase in the Na+-sensor neuronal population.”, J Neurosci, vol. 33, no. 7, pp. 3067-78, 2013.
, “Acidic residues on the voltage-sensor domain determine the activation of the NaChBac sodium channel.”, Biophys J, vol. 92, no. 10, pp. 3513-23, 2007.
, “Prolonged hyperpolarizing potentials precede spindle oscillations in the thalamic reticular nucleus.”, Proc Natl Acad Sci U S A, vol. 101, no. 26, pp. 9816-21, 2004.
, “Cortical hyperpolarization-activated depolarizing current takes part in the generation of focal paroxysmal activities.”, Proc Natl Acad Sci U S A, vol. 99, no. 14, pp. 9533-7, 2002.
, “Model of thalamocortical slow-wave sleep oscillations and transitions to activated States.”, J Neurosci, vol. 22, no. 19, pp. 8691-704, 2002.
, “Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).”, Circ Res, vol. 88, no. 12, pp. E78-83, 2001.
, “SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells.”, FEBS Lett, vol. 467, no. 1, pp. 12-6, 2000.
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