Publications
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, “Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide.”, FASEB J, vol. 31, no. 7, pp. 3066-3071, 2017.
, “Cumulus Cell Transcripts Transit to the Bovine Oocyte in Preparation for Maturation.”, Biol Reprod, vol. 94, no. 1, p. 16, 2016.
, “The gametic synapse: RNA transfer to the bovine oocyte.”, Biol Reprod, vol. 91, no. 4, p. 90, 2014.
, “Gating pore currents and the resting state of Nav1.4 voltage sensor domains.”, Proc Natl Acad Sci U S A, vol. 109, no. 47, pp. 19250-5, 2012.
, “A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype.”, PLoS One, vol. 7, no. 5, p. e38331, 2012.
, “Stoichiometry of the human glycine receptor revealed by direct subunit counting.”, J Neurosci, vol. 32, no. 37, pp. 12915-20, 2012.
, “Method to isolate polyribosomal mRNA from scarce samples such as mammalian oocytes and early embryos.”, BMC Dev Biol, vol. 11, p. 8, 2011.
, “Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine.”, Br J Pharmacol, vol. 142, no. 3, pp. 576-84, 2004.
, “Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C.”, J Neurophysiol, vol. 91, no. 4, pp. 1556-69, 2004.
, “A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels.”, Pflugers Arch, vol. 445, no. 1, pp. 18-24, 2002.
, “Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels.”, J Neurosci, vol. 21, no. 20, pp. 7909-18, 2001.
, “Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).”, Circ Res, vol. 88, no. 12, pp. E78-83, 2001.
, “SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells.”, FEBS Lett, vol. 467, no. 1, pp. 12-6, 2000.
, “Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site.”, Biophys J, vol. 75, no. 1, pp. 236-46, 1998.
, “Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels.”, FEBS Lett, vol. 412, no. 3, pp. 621-4, 1997.
, “Okadaic acid enhances prepulse facilitation of cardiac alpha 1-subunit but not endogenous calcium channel currents in Xenopus laevis oocytes.”, Can J Physiol Pharmacol, vol. 74, no. 10, pp. 1149-56, 1996.
, “Agonist and toxin sensitivities of ACh-evoked currents on neurons expressing multiple nicotinic ACh receptor subunits.”, J Neurophysiol, vol. 74, no. 3, pp. 1212-21, 1995.
, “Characterizing the mu-conotoxin binding site on voltage-sensitive sodium channels with toxin analogs and channel mutations.”, Receptors Channels, vol. 3, no. 3, pp. 161-74, 1995.
