Publications
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Filters: Keyword is Ion Channel Gating [Clear All Filters]
, “Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides.”, Sci Rep, vol. 5, p. 12475, 2015.
, “Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy.”, J Gen Physiol, vol. 145, no. 2, pp. 93-106, 2015.
, “Fluoxetine blocks Nav1.5 channels via a mechanism similar to that of class 1 antiarrhythmics.”, Mol Pharmacol, vol. 86, no. 4, pp. 378-89, 2014.
, “Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?”, Channels (Austin), vol. 8, no. 1, pp. 90-4, 2014.
, “Pyridoxal-5'-phosphate (MC-1), a vitamin B6 derivative, inhibits expressed P2X receptors.”, Can J Physiol Pharmacol, vol. 92, no. 3, pp. 189-96, 2014.
, “Gating pore currents and the resting state of Nav1.4 voltage sensor domains.”, Proc Natl Acad Sci U S A, vol. 109, no. 47, pp. 19250-5, 2012.
, “Simultaneous optical recording in multiple cells by digital holographic microscopy of chloride current associated to activation of the ligand-gated chloride channel GABA(A) receptor.”, PLoS One, vol. 7, no. 12, p. e51041, 2012.
, “Regulation of Nav1.6 and Nav1.8 peripheral nerve Na+ channels by auxiliary β-subunits.”, J Neurophysiol, vol. 106, no. 2, pp. 608-19, 2011.
, “Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels.”, Am J Physiol Heart Circ Physiol, vol. 300, no. 1, pp. H288-99, 2011.
, “Biophysical characterization of a new SCN5A mutation S1333Y in a SIDS infant linked to long QT syndrome.”, FEBS Lett, vol. 583, no. 5, pp. 890-6, 2009.
, “Protein kinase C activation inhibits alpha1D L-type Ca channel: a single-channel analysis.”, Pflugers Arch, vol. 455, no. 5, pp. 913-9, 2008.
, “Voltage-gated sodium channels in neurological disorders.”, CNS Neurol Disord Drug Targets, vol. 7, no. 2, pp. 144-58, 2008.
, “Accessibility of four arginine residues on the S4 segment of the Bacillus halodurans sodium channel.”, J Membr Biol, vol. 215, no. 2-3, pp. 169-80, 2007.
, “Acidic residues on the voltage-sensor domain determine the activation of the NaChBac sodium channel.”, Biophys J, vol. 92, no. 10, pp. 3513-23, 2007.
, “Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine.”, Br J Pharmacol, vol. 142, no. 3, pp. 576-84, 2004.
, “Role of arginine residues on the S4 segment of the Bacillus halodurans Na+ channel in voltage-sensing.”, J Membr Biol, vol. 201, no. 1, pp. 9-24, 2004.
, “Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels.”, J Neurosci, vol. 21, no. 20, pp. 7909-18, 2001.
, “Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation.”, J Membr Biol, vol. 183, no. 2, pp. 103-14, 2001.
, “Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).”, Circ Res, vol. 88, no. 12, pp. E78-83, 2001.
, “Cysteine scanning analysis of the IFM cluster in the inactivation gate of a human heart sodium channel.”, Cardiovasc Res, vol. 42, no. 2, pp. 521-9, 1999.
, “Electrophysiological study of chimeric sodium channels from heart and skeletal muscle.”, J Membr Biol, vol. 164, no. 1, pp. 25-34, 1998.
, “Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels.”, FEBS Lett, vol. 412, no. 3, pp. 621-4, 1997.
, “Restoration of fast inactivation in an inactivation-defective human heart sodium channel by the cysteine modifying reagent benzyl-MTS: analysis of IFM-ICM mutation.”, Biochem Biophys Res Commun, vol. 233, no. 3, pp. 606-10, 1997.
, “Sea anemone toxin (ATX II) modulation of heart and skeletal muscle sodium channel alpha-subunits expressed in tsA201 cells.”, J Membr Biol, vol. 152, no. 1, pp. 39-48, 1996.
