Publications
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Filters: Keyword is Sodium Channels and Author is Mohamed Chahine [Clear All Filters]
, “Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide.”, FASEB J, vol. 31, no. 7, pp. 3066-3071, 2017.
, “Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.”, Can J Cardiol, vol. 33, no. 2, pp. 269-278, 2017.
, “A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype.”, PLoS One, vol. 7, no. 5, p. e38331, 2012.
, “Regulation of Nav1.6 and Nav1.8 peripheral nerve Na+ channels by auxiliary β-subunits.”, J Neurophysiol, vol. 106, no. 2, pp. 608-19, 2011.
, “Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels.”, Am J Physiol Heart Circ Physiol, vol. 300, no. 1, pp. H288-99, 2011.
, “Cell membrane expression of cardiac sodium channel Na(v)1.5 is modulated by alpha-actinin-2 interaction.”, Biochemistry, vol. 49, no. 1, pp. 166-78, 2010.
, “Biophysical characterization of a new SCN5A mutation S1333Y in a SIDS infant linked to long QT syndrome.”, FEBS Lett, vol. 583, no. 5, pp. 890-6, 2009.
, “Biophysical properties of human Na v1.7 splice variants and their regulation by protein kinase A.”, J Neurophysiol, vol. 99, no. 5, pp. 2241-50, 2008.
, “Voltage-gated sodium channels in neurological disorders.”, CNS Neurol Disord Drug Targets, vol. 7, no. 2, pp. 144-58, 2008.
, “Accessibility of four arginine residues on the S4 segment of the Bacillus halodurans sodium channel.”, J Membr Biol, vol. 215, no. 2-3, pp. 169-80, 2007.
, “Acidic residues on the voltage-sensor domain determine the activation of the NaChBac sodium channel.”, Biophys J, vol. 92, no. 10, pp. 3513-23, 2007.
, “Lidocaine promotes the trafficking and functional expression of Na(v)1.8 sodium channels in mammalian cells.”, J Neurophysiol, vol. 98, no. 1, pp. 467-77, 2007.
, “Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.”, Can J Physiol Pharmacol, vol. 84, no. 8-9, pp. 795-802, 2006.
, “Nav1.5/R1193Q polymorphism is associated with both long QT and Brugada syndromes.”, Can J Cardiol, vol. 22, no. 4, pp. 309-13, 2006.
, “Regulation of Na v channels in sensory neurons.”, Trends Pharmacol Sci, vol. 26, no. 10, pp. 496-502, 2005.
, “The C-terminal region as a modulator of rNa(v)1.7 and rNa(v)1.8 expression levels.”, FEBS Lett, vol. 559, no. 1-3, pp. 39-44, 2004.
, “Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine.”, Br J Pharmacol, vol. 142, no. 3, pp. 576-84, 2004.
, “Loss of function associated with novel mutations of the SCN5A gene in patients with Brugada syndrome.”, Can J Cardiol, vol. 20, no. 4, pp. 425-30, 2004.
, “Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C.”, J Neurophysiol, vol. 91, no. 4, pp. 1556-69, 2004.
, “Role of arginine residues on the S4 segment of the Bacillus halodurans Na+ channel in voltage-sensing.”, J Membr Biol, vol. 201, no. 1, pp. 9-24, 2004.
, “Role of auxiliary beta1-, beta2-, and beta3-subunits and their interaction with Na(v)1.8 voltage-gated sodium channel.”, Biochem Biophys Res Commun, vol. 319, no. 2, pp. 531-40, 2004.
, “Expression and intracellular localization of an SCN5A double mutant R1232W/T1620M implicated in Brugada syndrome.”, Circ Res, vol. 90, no. 1, pp. E11-6, 2002.
, “A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels.”, Pflugers Arch, vol. 445, no. 1, pp. 18-24, 2002.
, “Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels.”, J Neurosci, vol. 21, no. 20, pp. 7909-18, 2001.
, “Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation.”, J Membr Biol, vol. 183, no. 2, pp. 103-14, 2001.
