Publications
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Filters: Author is Thériault, Olivier [Clear All Filters]
“-related epilepsy of infancy with migrating focal seizures: report of a variant with apparent gain- and loss-of-function effects.”, J Neurophysiol, vol. 127, no. 5, pp. 1388-1397, 2022.
, “Novel G1481V and Q1491H SCN5A Mutations Linked to Long QT Syndrome Destabilize the Nav1.5 Inactivation State.”, CJC Open, vol. 3, no. 3, pp. 256-266, 2021.
, “Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.”, Can J Cardiol, vol. 33, no. 2, pp. 269-278, 2017.
, “Differential modulation of Nav1.7 and Nav1.8 channels by antidepressant drugs.”, Eur J Pharmacol, vol. 764, pp. 395-403, 2015.
, “Correlation of the electrophysiological profiles and sodium channel transcripts of individual rat dorsal root ganglia neurons.”, Front Cell Neurosci, vol. 8, p. 285, 2014.
, “Fluoxetine blocks Nav1.5 channels via a mechanism similar to that of class 1 antiarrhythmics.”, Mol Pharmacol, vol. 86, no. 4, pp. 378-89, 2014.
, “Modulation of peripheral Na(+) channels and neuronal firing by n-butyl-p-aminobenzoate.”, Eur J Pharmacol, vol. 727, pp. 158-66, 2014.
, “Pyridoxal-5'-phosphate (MC-1), a vitamin B6 derivative, inhibits expressed P2X receptors.”, Can J Physiol Pharmacol, vol. 92, no. 3, pp. 189-96, 2014.
, “Coexisting mutations/polymorphisms of the long QT syndrome genes in patients with repaired Tetralogy of Fallot are associated with the risks of life-threatening events.”, Hum Genet, vol. 131, no. 8, pp. 1295-304, 2012.
, “A distinct de novo expression of Nav1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts.”, J Physiol, vol. 590, no. 17, pp. 4307-19, 2012.
, “In utero onset of long QT syndrome with atrioventricular block and spontaneous or lidocaine-induced ventricular tachycardia: compound effects of hERG pore region mutation and SCN5A N-terminus variant.”, Heart Rhythm, vol. 5, no. 11, pp. 1567-74, 2008.
, “A new C-terminal hERG mutation A915fs+47X associated with symptomatic LQT2 and auditory-trigger syncope.”, Heart Rhythm, vol. 5, no. 11, pp. 1577-86, 2008.
