Publications
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Filters: Author is Mohamed Chahine [Clear All Filters]
, “A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy.”, Sci Rep, vol. 8, no. 1, p. 13804, 2018.
, “A New Cardiac Channelopathy: From Clinical Phenotypes to Molecular Mechanisms Associated With Na1.5 Gating Pores.”, Front Cardiovasc Med, vol. 5, p. 139, 2018.
, “Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide.”, FASEB J, vol. 31, no. 7, pp. 3066-3071, 2017.
, “Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.”, Can J Cardiol, vol. 33, no. 2, pp. 269-278, 2017.
, “Induced pluripotent stem-cell-derived cardiomyocytes: cardiac applications, opportunities, and challenges.”, Can J Physiol Pharmacol, vol. 95, no. 10, pp. 1108-1116, 2017.
, “Mechanisms of Drug Binding to Voltage-Gated Sodium Channels.”, Handb Exp Pharmacol, 2017.
, “Metaflumizone inhibits the honeybee NaV 1 channel by targeting recovery from slow inactivation.”, FEBS Lett, 2017.
, “Regulation of Cardiac Voltage-Gated Sodium Channel by Kinases: Roles of Protein Kinases A and C.”, Handb Exp Pharmacol, 2017.
, “Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4.”, J Gen Physiol, vol. 148, no. 2, pp. 133-45, 2016.
, “Editorial: Recent Advances in Voltage-Gated Sodium Channels, their Pharmacology and Related Diseases.”, Front Pharmacol, vol. 7, p. 20, 2016.
, “Induction of autoimmune response to the extracellular loop of the HERG channel pore induces QTc prolongation in guinea-pigs.”, J Physiol, vol. 594, no. 21, pp. 6175-6187, 2016.
, “A recessive Nav1.4 mutation underlies congenital myasthenic syndrome with periodic paralysis.”, Neurology, vol. 86, no. 2, pp. 161-9, 2016.
, “Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides.”, Sci Rep, vol. 5, p. 12475, 2015.
, “Differential modulation of Nav1.7 and Nav1.8 channels by antidepressant drugs.”, Eur J Pharmacol, vol. 764, pp. 395-403, 2015.
, “Effects of amlodipine and perindoprilate on the structure and function of mitochondria in ventricular cardiomyocytes during ischemia-reperfusion in the pig.”, Fundam Clin Pharmacol, vol. 29, no. 1, pp. 21-30, 2015.
, “Gating pore current is a novel biophysical defect of Nav1.5 mutations associated with unusual cardiac arrhythmias and dilation.”, Future Cardiol, vol. 11, no. 3, pp. 287-91, 2015.
, “Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy.”, Channels (Austin), vol. 9, no. 3, pp. 139-44, 2015.
, “Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy.”, J Gen Physiol, vol. 145, no. 2, pp. 93-106, 2015.
, “Molecular characterization and functional expression of the Apis mellifera voltage-dependent Ca2+ channels.”, Insect Biochem Mol Biol, vol. 58, pp. 12-27, 2015.
, “MTSET modification of D4S6 cysteines stabilize the fast inactivated state of Nav1.5 sodium channels.”, Front Pharmacol, vol. 6, p. 118, 2015.
, “Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents.”, Front Pharmacol, vol. 6, p. 301, 2015.
, “[Omega pore, an alternative ion channel permeation pathway involved in the development of several channelopathies].”, Med Sci (Paris), vol. 31, no. 8-9, pp. 735-41, 2015.
, “Pathogenesis of the Novel Autoimmune-Associated Long-QT Syndrome.”, Circulation, vol. 132, no. 4, pp. 230-40, 2015.
, “Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1.”, Ann Noninvasive Electrocardiol, vol. 20, no. 1, pp. 28-36, 2015.
, “Biophysics, pathophysiology, and pharmacology of ion channel gating pores.”, Front Pharmacol, vol. 5, p. 53, 2014.
