Publications
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, “Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1.”, Ann Noninvasive Electrocardiol, vol. 20, no. 1, pp. 28-36, 2015.
, “Myotonic dystrophy type 1 mimics and exacerbates Brugada phenotype induced by Nav1.5 sodium channel loss-of-function mutation.”, Heart Rhythm, vol. 11, no. 8, pp. 1393-400, 2014.
, “A distinct de novo expression of Nav1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts.”, J Physiol, vol. 590, no. 17, pp. 4307-19, 2012.
, “A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype.”, PLoS One, vol. 7, no. 5, p. e38331, 2012.
, “The β1-subunit of Na(v)1.5 cardiac sodium channel is required for a dominant negative effect through α-α interaction.”, PLoS One, vol. 7, no. 11, p. e48690, 2012.
, “Biophysical characterisation of the persistent sodium current of the Nav1.6 neuronal sodium channel: a single-channel analysis.”, Pflugers Arch, vol. 460, no. 1, pp. 77-86, 2010.
, “Biophysical properties of human Na v1.7 splice variants and their regulation by protein kinase A.”, J Neurophysiol, vol. 99, no. 5, pp. 2241-50, 2008.
, “Voltage-gated sodium channels in neurological disorders.”, CNS Neurol Disord Drug Targets, vol. 7, no. 2, pp. 144-58, 2008.
, “Lidocaine promotes the trafficking and functional expression of Na(v)1.8 sodium channels in mammalian cells.”, J Neurophysiol, vol. 98, no. 1, pp. 467-77, 2007.
