Publications
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, “A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels.”, Pflugers Arch, vol. 445, no. 1, pp. 18-24, 2002.
, “Ethanol delays and reverses lysophosphatidylcholine-induced calcium overload in neonatal rat heart cells.”, Pflugers Arch, vol. 443, no. 1, pp. 48-53, 2001.
, “Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels.”, J Neurosci, vol. 21, no. 20, pp. 7909-18, 2001.
, “Homozygous SCN5A mutation in long-QT syndrome with functional two-to-one atrioventricular block.”, Circ Res, vol. 89, no. 2, pp. E16-21, 2001.
, “Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation.”, J Membr Biol, vol. 183, no. 2, pp. 103-14, 2001.
, “Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G).”, Circ Res, vol. 88, no. 12, pp. E78-83, 2001.
, “Biophysical phenotypes of SCN5A mutations causing long QT and Brugada syndromes.”, FEBS Lett, vol. 487, no. 2, pp. 224-8, 2000.
, “The distinct HERG missense mutation L564P causes long QT syndrome in one French Canadian family.”, Can J Cardiol, vol. 16, no. 3, pp. 307-12, 2000.
, “Electrophysiological characterization of SCN5A mutations causing long QT (E1784K) and Brugada (R1512W and R1432G) syndromes.”, Cardiovasc Res, vol. 46, no. 1, pp. 55-65, 2000.
, “SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells.”, FEBS Lett, vol. 467, no. 1, pp. 12-6, 2000.
, “Cysteine scanning analysis of the IFM cluster in the inactivation gate of a human heart sodium channel.”, Cardiovasc Res, vol. 42, no. 2, pp. 521-9, 1999.
, “Modulation of HERG potassium channel properties by external pH.”, Pflugers Arch, vol. 438, no. 3, pp. 419-22, 1999.
, “Thioridazine lengthens repolarization of cardiac ventricular myocytes by blocking the delayed rectifier potassium current.”, J Pharmacol Exp Ther, vol. 288, no. 3, pp. 1261-8, 1999.
, “Electrophysiological study of chimeric sodium channels from heart and skeletal muscle.”, J Membr Biol, vol. 164, no. 1, pp. 25-34, 1998.
, “Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site.”, Biophys J, vol. 75, no. 1, pp. 236-46, 1998.
, “Effects of Tityus serrulatus scorpion toxin gamma on voltage-gated Na+ channels.”, Circ Res, vol. 80, no. 3, pp. 363-9, 1997.
, “Increase in the proliferative capacity of human myoblasts by using the T antigen under the vimentin promoter control.”, Muscle Nerve, vol. 20, no. 4, pp. 437-45, 1997.
, “Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels.”, FEBS Lett, vol. 412, no. 3, pp. 621-4, 1997.
, “Restoration of fast inactivation in an inactivation-defective human heart sodium channel by the cysteine modifying reagent benzyl-MTS: analysis of IFM-ICM mutation.”, Biochem Biophys Res Commun, vol. 233, no. 3, pp. 606-10, 1997.
, “Electrophysiological characteristics of cloned skeletal and cardiac muscle sodium channels.”, Am J Physiol, vol. 271, no. 2 Pt 2, pp. H498-506, 1996.
, “Okadaic acid enhances prepulse facilitation of cardiac alpha 1-subunit but not endogenous calcium channel currents in Xenopus laevis oocytes.”, Can J Physiol Pharmacol, vol. 74, no. 10, pp. 1149-56, 1996.
, “Sea anemone toxin (ATX II) modulation of heart and skeletal muscle sodium channel alpha-subunits expressed in tsA201 cells.”, J Membr Biol, vol. 152, no. 1, pp. 39-48, 1996.
, “Characterizing the mu-conotoxin binding site on voltage-sensitive sodium channels with toxin analogs and channel mutations.”, Receptors Channels, vol. 3, no. 3, pp. 161-74, 1995.
, “Functional expression and properties of the human skeletal muscle sodium channel.”, Pflugers Arch, vol. 427, no. 1-2, pp. 136-42, 1994.
, “Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation.”, Neuron, vol. 12, no. 2, pp. 281-94, 1994.
