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Filtres: Author is Mohamed Chahine [Enlever les filtres]
Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide. FASEB J. 2017;31(7):3066-3071.
Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes. Can J Cardiol. 2017;33(2):269-278.
Induced pluripotent stem-cell-derived cardiomyocytes: cardiac applications, opportunities, and challenges. Can J Physiol Pharmacol. 2017;95(10):1108-1116.
Mechanisms of Drug Binding to Voltage-Gated Sodium Channels. Handb Exp Pharmacol. 2017.
Metaflumizone inhibits the honeybee NaV 1 channel by targeting recovery from slow inactivation. FEBS Lett. 2017.
Regulation of Cardiac Voltage-Gated Sodium Channel by Kinases: Roles of Protein Kinases A and C. Handb Exp Pharmacol. 2017.
Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4. J Gen Physiol. 2016;148(2):133-45.
Editorial: Recent Advances in Voltage-Gated Sodium Channels, their Pharmacology and Related Diseases. Front Pharmacol. 2016;7:20.
Induction of autoimmune response to the extracellular loop of the HERG channel pore induces QTc prolongation in guinea-pigs. J Physiol (Lond). 2016;594(21):6175-6187.
A recessive Nav1.4 mutation underlies congenital myasthenic syndrome with periodic paralysis. Neurology. 2016;86(2):161-9.
Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides. Sci Rep. 2015;5:12475.
Differential modulation of Nav1.7 and Nav1.8 channels by antidepressant drugs. Eur J Pharmacol. 2015;764:395-403.
Effects of amlodipine and perindoprilate on the structure and function of mitochondria in ventricular cardiomyocytes during ischemia-reperfusion in the pig. Fundam Clin Pharmacol. 2015;29(1):21-30.
Gating pore current is a novel biophysical defect of Nav1.5 mutations associated with unusual cardiac arrhythmias and dilation. Future Cardiol. 2015;11(3):287-91.
Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy. Channels (Austin). 2015;9(3):139-44.
Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy. J Gen Physiol. 2015;145(2):93-106.
Molecular characterization and functional expression of the Apis mellifera voltage-dependent Ca2+ channels. Insect Biochem Mol Biol. 2015;58:12-27.
MTSET modification of D4S6 cysteines stabilize the fast inactivated state of Nav1.5 sodium channels. Front Pharmacol. 2015;6:118.
Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents. Front Pharmacol. 2015;6:301.
[Omega pore, an alternative ion channel permeation pathway involved in the development of several channelopathies]. Med Sci (Paris). 2015;31(8-9):735-41.
Pathogenesis of the Novel Autoimmune-Associated Long-QT Syndrome. Circulation. 2015;132(4):230-40.
Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1. Ann Noninvasive Electrocardiol. 2015;20(1):28-36.
Biophysics, pathophysiology, and pharmacology of ion channel gating pores. Front Pharmacol. 2014;5:53.
Correlation of the electrophysiological profiles and sodium channel transcripts of individual rat dorsal root ganglia neurons. Front Cell Neurosci. 2014;8:285.
Fluoxetine blocks Nav1.5 channels via a mechanism similar to that of class 1 antiarrhythmics. Mol Pharmacol. 2014;86(4):378-89.