Publications
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Filtres: Author is Gosselin-Badaroudine, Pascal [Enlever les filtres]
« A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy. », Sci Rep, vol. 8, nᵒ 1, p. 13804, 2018.
, « Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide. », FASEB J, vol. 31, nᵒ 7, p. 3066-3071, 2017.
, , « Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4. », J Gen Physiol, vol. 148, nᵒ 2, p. 133-45, 2016.
, « Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides. », Sci Rep, vol. 5, p. 12475, 2015.
, « Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy. », Channels (Austin), vol. 9, nᵒ 3, p. 139-44, 2015.
, « Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy. », J Gen Physiol, vol. 145, nᵒ 2, p. 93-106, 2015.
, , « Biophysics, pathophysiology, and pharmacology of ion channel gating pores. », Front Pharmacol, vol. 5, p. 53, 2014.
, « Molecular biology and biophysical properties of ion channel gating pores. », Q Rev Biophys, vol. 47, nᵒ 4, p. 364-88, 2014.
, « Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link? », Channels (Austin), vol. 8, nᵒ 1, p. 90-4, 2014.
, « Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutation. », Front Pharmacol, vol. 4, p. 126, 2013.
, « Gating pore currents and the resting state of Nav1.4 voltage sensor domains. », Proc Natl Acad Sci U S A, vol. 109, nᵒ 47, p. 19250-5, 2012.
, « A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype. », PLoS One, vol. 7, nᵒ 5, p. e38331, 2012.
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