Publications
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Filtres: Keyword is Phosphorylation [Enlever les filtres]
« The Tyrosine Phosphatase STEP Is Involved in Age-Related Memory Decline. », Curr Biol, vol. 28, nᵒ 7, p. 1079-1089.e4, 2018.
, « New Phosphospecific Antibody Reveals Isoform-Specific Phosphorylation of CPEB3 Protein. », PLoS One, vol. 11, nᵒ 2, p. e0150000, 2016.
, « Interaction between αCaMKII and GluN2B controls ERK-dependent plasticity. », J Neurosci, vol. 32, nᵒ 31, p. 10767-79, 2012.
, « Translocation of CaMKII to dendritic microtubules supports the plasticity of local synapses. », J Cell Biol, vol. 198, nᵒ 6, p. 1055-73, 2012.
, « Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation. », J Neurosci, vol. 30, nᵒ 26, p. 8704-9, 2010.
, « CaMKII control of spine size and synaptic strength: role of phosphorylation states and nonenzymatic action. », Proc Natl Acad Sci U S A, vol. 107, nᵒ 32, p. 14437-42, 2010.
, « CaMKII triggers the diffusional trapping of surface AMPARs through phosphorylation of stargazin. », Neuron, vol. 67, nᵒ 2, p. 239-52, 2010.
, « Biophysical properties of human Na v1.7 splice variants and their regulation by protein kinase A. », J Neurophysiol, vol. 99, nᵒ 5, p. 2241-50, 2008.
, « Haloperidol induces the nuclear translocation of phosphatidylinositol 3'-kinase to disrupt Akt phosphorylation in PC12 cells. », J Psychiatry Neurosci, vol. 32, nᵒ 5, p. 323-30, 2007.
, « Aggregate formation and phosphorylation of neurofilament-L Pro22 Charcot-Marie-Tooth disease mutants. », Hum Mol Genet, vol. 15, nᵒ 6, p. 943-52, 2006.
, « Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B. », J Neurosci, vol. 26, nᵒ 4, p. 1164-74, 2006.
, « The high and middle molecular weight neurofilament subunits regulate the association of neurofilaments with kinesin: inhibition by phosphorylation of the high molecular weight subunit. », Brain Res Mol Brain Res, vol. 141, nᵒ 2, p. 151-5, 2005.
, « A mechanism for Ca2+/calmodulin-dependent protein kinase II clustering at synaptic and nonsynaptic sites based on self-association. », J Neurosci, vol. 25, nᵒ 30, p. 6971-83, 2005.
, « Cell cycle regulators in the neuronal death pathway of amyotrophic lateral sclerosis caused by mutant superoxide dismutase 1. », J Neurosci, vol. 23, nᵒ 6, p. 2131-40, 2003.
, « Cyclin-dependent kinase 5 in amyotrophic lateral sclerosis. », Neurosignals, vol. 12, nᵒ 4-5, p. 215-20, 2003.
, « Fragile X Mental Retardation protein determinants required for its association with polyribosomal mRNPs. », Hum Mol Genet, vol. 12, nᵒ 23, p. 3087-96, 2003.
, « Sensitivity of CaM kinase II to the frequency of Ca2+ oscillations: a simple model. », Cell Calcium, vol. 34, nᵒ 6, p. 485-97, 2003.
, « Alternative splicing modulates the frequency-dependent response of CaMKII to Ca(2+) oscillations. », EMBO J, vol. 21, nᵒ 14, p. 3590-7, 2002.
, « Interaction with the NMDA receptor locks CaMKII in an active conformation. », Nature, vol. 411, nᵒ 6839, p. 801-5, 2001.
, « Sensitivity of CaM kinase II to the frequency of Ca2+ oscillations. », Science, vol. 279, nᵒ 5348, p. 227-30, 1998.
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