Publications
Export 158 results:
Filtres: Author is Mohamed Chahine [Enlever les filtres]
, « A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy. », Sci Rep, vol. 8, nᵒ 1, p. 13804, 2018.
, « A New Cardiac Channelopathy: From Clinical Phenotypes to Molecular Mechanisms Associated With Na1.5 Gating Pores. », Front Cardiovasc Med, vol. 5, p. 139, 2018.
, « Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ-fluvalinate insecticide. », FASEB J, vol. 31, nᵒ 7, p. 3066-3071, 2017.
, « Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes. », Can J Cardiol, vol. 33, nᵒ 2, p. 269-278, 2017.
, « Induced pluripotent stem-cell-derived cardiomyocytes: cardiac applications, opportunities, and challenges. », Can J Physiol Pharmacol, vol. 95, nᵒ 10, p. 1108-1116, 2017.
, « Mechanisms of Drug Binding to Voltage-Gated Sodium Channels. », Handb Exp Pharmacol, 2017.
, « Metaflumizone inhibits the honeybee NaV 1 channel by targeting recovery from slow inactivation. », FEBS Lett, 2017.
, « Regulation of Cardiac Voltage-Gated Sodium Channel by Kinases: Roles of Protein Kinases A and C. », Handb Exp Pharmacol, 2017.
, « Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4. », J Gen Physiol, vol. 148, nᵒ 2, p. 133-45, 2016.
, « Editorial: Recent Advances in Voltage-Gated Sodium Channels, their Pharmacology and Related Diseases. », Front Pharmacol, vol. 7, p. 20, 2016.
, « Induction of autoimmune response to the extracellular loop of the HERG channel pore induces QTc prolongation in guinea-pigs. », J Physiol, vol. 594, nᵒ 21, p. 6175-6187, 2016.
, « A recessive Nav1.4 mutation underlies congenital myasthenic syndrome with periodic paralysis. », Neurology, vol. 86, nᵒ 2, p. 161-9, 2016.
, « Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides. », Sci Rep, vol. 5, p. 12475, 2015.
, « Differential modulation of Nav1.7 and Nav1.8 channels by antidepressant drugs. », Eur J Pharmacol, vol. 764, p. 395-403, 2015.
, « Effects of amlodipine and perindoprilate on the structure and function of mitochondria in ventricular cardiomyocytes during ischemia-reperfusion in the pig. », Fundam Clin Pharmacol, vol. 29, nᵒ 1, p. 21-30, 2015.
, « Gating pore current is a novel biophysical defect of Nav1.5 mutations associated with unusual cardiac arrhythmias and dilation. », Future Cardiol, vol. 11, nᵒ 3, p. 287-91, 2015.
, « Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy. », Channels (Austin), vol. 9, nᵒ 3, p. 139-44, 2015.
, « Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy. », J Gen Physiol, vol. 145, nᵒ 2, p. 93-106, 2015.
, « Molecular characterization and functional expression of the Apis mellifera voltage-dependent Ca2+ channels. », Insect Biochem Mol Biol, vol. 58, p. 12-27, 2015.
, « MTSET modification of D4S6 cysteines stabilize the fast inactivated state of Nav1.5 sodium channels. », Front Pharmacol, vol. 6, p. 118, 2015.
, « Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents. », Front Pharmacol, vol. 6, p. 301, 2015.
, « [Omega pore, an alternative ion channel permeation pathway involved in the development of several channelopathies]. », Med Sci (Paris), vol. 31, nᵒ 8-9, p. 735-41, 2015.
, « Pathogenesis of the Novel Autoimmune-Associated Long-QT Syndrome. », Circulation, vol. 132, nᵒ 4, p. 230-40, 2015.
, « Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1. », Ann Noninvasive Electrocardiol, vol. 20, nᵒ 1, p. 28-36, 2015.
, « Biophysics, pathophysiology, and pharmacology of ion channel gating pores. », Front Pharmacol, vol. 5, p. 53, 2014.
