Publications
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Filtres: Author is Mohamed Chahine [Enlever les filtres]
, « Molecular characterization and functional expression of the Apis mellifera voltage-dependent Ca2+ channels. », Insect Biochem Mol Biol, vol. 58, p. 12-27, 2015.
, « MTSET modification of D4S6 cysteines stabilize the fast inactivated state of Nav1.5 sodium channels. », Front Pharmacol, vol. 6, p. 118, 2015.
, « Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents. », Front Pharmacol, vol. 6, p. 301, 2015.
, « [Omega pore, an alternative ion channel permeation pathway involved in the development of several channelopathies]. », Med Sci (Paris), vol. 31, nᵒ 8-9, p. 735-41, 2015.
, « Pathogenesis of the Novel Autoimmune-Associated Long-QT Syndrome. », Circulation, vol. 132, nᵒ 4, p. 230-40, 2015.
, « Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1. », Ann Noninvasive Electrocardiol, vol. 20, nᵒ 1, p. 28-36, 2015.
, « Biophysics, pathophysiology, and pharmacology of ion channel gating pores. », Front Pharmacol, vol. 5, p. 53, 2014.
, « Correlation of the electrophysiological profiles and sodium channel transcripts of individual rat dorsal root ganglia neurons. », Front Cell Neurosci, vol. 8, p. 285, 2014.
, « Fluoxetine blocks Nav1.5 channels via a mechanism similar to that of class 1 antiarrhythmics. », Mol Pharmacol, vol. 86, nᵒ 4, p. 378-89, 2014.
, « Modulation of peripheral Na(+) channels and neuronal firing by n-butyl-p-aminobenzoate. », Eur J Pharmacol, vol. 727, p. 158-66, 2014.
, « Molecular biology and biophysical properties of ion channel gating pores. », Q Rev Biophys, vol. 47, nᵒ 4, p. 364-88, 2014.
, « Myotonic dystrophy type 1 mimics and exacerbates Brugada phenotype induced by Nav1.5 sodium channel loss-of-function mutation. », Heart Rhythm, vol. 11, nᵒ 8, p. 1393-400, 2014.
, « Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link? », Channels (Austin), vol. 8, nᵒ 1, p. 90-4, 2014.
, « Pyrethroids differentially alter voltage-gated sodium channels from the honeybee central olfactory neurons. », PLoS One, vol. 9, nᵒ 11, p. e112194, 2014.
, « Pyridoxal-5'-phosphate (MC-1), a vitamin B6 derivative, inhibits expressed P2X receptors. », Can J Physiol Pharmacol, vol. 92, nᵒ 3, p. 189-96, 2014.
, « Regulation/modulation of sensory neuron sodium channels. », Handb Exp Pharmacol, vol. 221, p. 111-35, 2014.
, « The variant hERG/R148W associated with LQTS is a mutation that reduces current density on co-expression with the WT. », Gene, vol. 536, nᵒ 2, p. 348-56, 2014.
, « Characterization of the first honeybee Ca²⁺ channel subunit reveals two novel species- and splicing-specific modes of regulation of channel inactivation. », Pflugers Arch, vol. 465, nᵒ 7, p. 985-96, 2013.
, « Novel SCN5A mutations in two families with "Brugada-like" ST elevation in the inferior leads and conduction disturbances. », J Interv Card Electrophysiol, vol. 37, nᵒ 2, p. 131-40, 2013.
, « Recent advances in voltage-gated sodium channels, their pharmacology, and related diseases. », Front Pharmacol, vol. 4, p. 52, 2013.
, « Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutation. », Front Pharmacol, vol. 4, p. 126, 2013.
, « Biophysical characterization of M1476I, a sodium channel founder mutation associated with cold-induced myotonia in French Canadians. », J Physiol, vol. 590, nᵒ 11, p. 2629-44, 2012.
, « Coexisting mutations/polymorphisms of the long QT syndrome genes in patients with repaired Tetralogy of Fallot are associated with the risks of life-threatening events. », Hum Genet, vol. 131, nᵒ 8, p. 1295-304, 2012.
, « Differential expression of sodium channel β subunits in dorsal root ganglion sensory neurons. », J Biol Chem, vol. 287, nᵒ 18, p. 15044-53, 2012.
, « A distinct de novo expression of Nav1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts. », J Physiol, vol. 590, nᵒ 17, p. 4307-19, 2012.
