Publications
Export 158 results:
Filtres: Author is Mohamed Chahine [Enlever les filtres]
, « A novel mutation in the SCN5A gene is associated with Brugada syndrome. », Life Sci, vol. 80, nᵒ 8, p. 716-24, 2007.
, « [A channel without pore? The primary structure of a proton permeable channel is finally revealed]. », Med Sci (Paris), vol. 22, nᵒ 11, p. 930-1, 2006.
, « Clinical aspects and physiopathology of Brugada syndrome: review of current concepts. », Can J Physiol Pharmacol, vol. 84, nᵒ 8-9, p. 795-802, 2006.
, « Nav1.5/R1193Q polymorphism is associated with both long QT and Brugada syndromes. », Can J Cardiol, vol. 22, nᵒ 4, p. 309-13, 2006.
, « A novel SCN5A mutation, F1344S, identified in a patient with Brugada syndrome and fever-induced ventricular fibrillation. », Cardiovasc Res, vol. 70, nᵒ 3, p. 521-9, 2006.
, « Protein kinase C activation inhibits Cav1.3 calcium channel at NH2-terminal serine 81 phosphorylation site. », Am J Physiol Heart Circ Physiol, vol. 291, nᵒ 4, p. H1614-22, 2006.
, « SCN5A polymorphism restores trafficking of a Brugada syndrome mutation on a separate gene. », Circulation, vol. 114, nᵒ 5, p. 368-76, 2006.
, « A novel nonsense mutation in the SCN5A gene leads to Brugada syndrome and a silent gene mutation carrier state. », Can J Cardiol, vol. 21, nᵒ 11, p. 925-31, 2005.
, « Regulation of Na v channels in sensory neurons. », Trends Pharmacol Sci, vol. 26, nᵒ 10, p. 496-502, 2005.
, « The C-terminal region as a modulator of rNa(v)1.7 and rNa(v)1.8 expression levels. », FEBS Lett, vol. 559, nᵒ 1-3, p. 39-44, 2004.
, « Differential modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by the local anesthetic lidocaine. », Br J Pharmacol, vol. 142, nᵒ 3, p. 576-84, 2004.
, « Genetic analysis of the cardiac sodium channel gene SCN5A in Koreans with Brugada syndrome. », J Hum Genet, vol. 49, nᵒ 10, p. 573-8, 2004.
, « Loss of function associated with novel mutations of the SCN5A gene in patients with Brugada syndrome. », Can J Cardiol, vol. 20, nᵒ 4, p. 425-30, 2004.
, « Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C. », J Neurophysiol, vol. 91, nᵒ 4, p. 1556-69, 2004.
, « A novel SCN5A mutation manifests as a malignant form of long QT syndrome with perinatal onset of tachycardia/bradycardia. », Cardiovasc Res, vol. 64, nᵒ 2, p. 268-78, 2004.
, « Role of arginine residues on the S4 segment of the Bacillus halodurans Na+ channel in voltage-sensing. », J Membr Biol, vol. 201, nᵒ 1, p. 9-24, 2004.
, « Role of auxiliary beta1-, beta2-, and beta3-subunits and their interaction with Na(v)1.8 voltage-gated sodium channel. », Biochem Biophys Res Commun, vol. 319, nᵒ 2, p. 531-40, 2004.
, « Biophysical characteristics of a new mutation on the KCNQ1 potassium channel (L251P) causing long QT syndrome. », Can J Physiol Pharmacol, vol. 81, nᵒ 2, p. 129-34, 2003.
, « Closing and inactivation potentiate the cocaethylene inhibition of cardiac sodium channels by distinct mechanisms. », Mol Pharmacol, vol. 64, nᵒ 6, p. 1575-85, 2003.
, « Modulation of L-type Ca2+ channels in neonatal rat heart by a novel Ca2+ channel agonist. », Can J Physiol Pharmacol, vol. 81, nᵒ 2, p. 135-41, 2003.
, « A newly characterized SCN5A mutation underlying Brugada syndrome unmasked by hyperthermia. », J Cardiovasc Electrophysiol, vol. 14, nᵒ 4, p. 407-11, 2003.
, « A novel mutation in SCN5A, delQKP 1507-1509, causing long QT syndrome: role of Q1507 residue in sodium channel inactivation. », J Mol Cell Cardiol, vol. 35, nᵒ 12, p. 1513-21, 2003.
, « Pathophysiology of Brugada syndrome. », J Cardiovasc Electrophysiol, vol. 14, nᵒ 11, p. 1257-8; author reply 1258, 2003.
, « Cocaine binds to a common site on open and inactivated human heart (Na(v)1.5) sodium channels. », J Physiol, vol. 541, nᵒ Pt 3, p. 701-16, 2002.
, « Expression and intracellular localization of an SCN5A double mutant R1232W/T1620M implicated in Brugada syndrome. », Circ Res, vol. 90, nᵒ 1, p. E11-6, 2002.
