Publications
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Filtres: Author is Mohamed Chahine [Enlever les filtres]
, « Closing and inactivation potentiate the cocaethylene inhibition of cardiac sodium channels by distinct mechanisms. », Mol Pharmacol, vol. 64, nᵒ 6, p. 1575-85, 2003.
, « Modulation of L-type Ca2+ channels in neonatal rat heart by a novel Ca2+ channel agonist. », Can J Physiol Pharmacol, vol. 81, nᵒ 2, p. 135-41, 2003.
, « A newly characterized SCN5A mutation underlying Brugada syndrome unmasked by hyperthermia. », J Cardiovasc Electrophysiol, vol. 14, nᵒ 4, p. 407-11, 2003.
, « A novel mutation in SCN5A, delQKP 1507-1509, causing long QT syndrome: role of Q1507 residue in sodium channel inactivation. », J Mol Cell Cardiol, vol. 35, nᵒ 12, p. 1513-21, 2003.
, « Pathophysiology of Brugada syndrome. », J Cardiovasc Electrophysiol, vol. 14, nᵒ 11, p. 1257-8; author reply 1258, 2003.
, « Cocaine binds to a common site on open and inactivated human heart (Na(v)1.5) sodium channels. », J Physiol, vol. 541, nᵒ Pt 3, p. 701-16, 2002.
, « Expression and intracellular localization of an SCN5A double mutant R1232W/T1620M implicated in Brugada syndrome. », Circ Res, vol. 90, nᵒ 1, p. E11-6, 2002.
, « A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels. », Pflugers Arch, vol. 445, nᵒ 1, p. 18-24, 2002.
, « Ethanol delays and reverses lysophosphatidylcholine-induced calcium overload in neonatal rat heart cells. », Pflugers Arch, vol. 443, nᵒ 1, p. 48-53, 2001.
, « Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels. », J Neurosci, vol. 21, nᵒ 20, p. 7909-18, 2001.
, « Homozygous SCN5A mutation in long-QT syndrome with functional two-to-one atrioventricular block. », Circ Res, vol. 89, nᵒ 2, p. E16-21, 2001.
, « Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation. », J Membr Biol, vol. 183, nᵒ 2, p. 103-14, 2001.
, « Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G). », Circ Res, vol. 88, nᵒ 12, p. E78-83, 2001.
, « Biophysical phenotypes of SCN5A mutations causing long QT and Brugada syndromes. », FEBS Lett, vol. 487, nᵒ 2, p. 224-8, 2000.
, « The distinct HERG missense mutation L564P causes long QT syndrome in one French Canadian family. », Can J Cardiol, vol. 16, nᵒ 3, p. 307-12, 2000.
, « Electrophysiological characterization of SCN5A mutations causing long QT (E1784K) and Brugada (R1512W and R1432G) syndromes. », Cardiovasc Res, vol. 46, nᵒ 1, p. 55-65, 2000.
, « SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells. », FEBS Lett, vol. 467, nᵒ 1, p. 12-6, 2000.
, « Cysteine scanning analysis of the IFM cluster in the inactivation gate of a human heart sodium channel. », Cardiovasc Res, vol. 42, nᵒ 2, p. 521-9, 1999.
, « Modulation of HERG potassium channel properties by external pH. », Pflugers Arch, vol. 438, nᵒ 3, p. 419-22, 1999.
, « Thioridazine lengthens repolarization of cardiac ventricular myocytes by blocking the delayed rectifier potassium current. », J Pharmacol Exp Ther, vol. 288, nᵒ 3, p. 1261-8, 1999.
, « Electrophysiological study of chimeric sodium channels from heart and skeletal muscle. », J Membr Biol, vol. 164, nᵒ 1, p. 25-34, 1998.
, « Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site. », Biophys J, vol. 75, nᵒ 1, p. 236-46, 1998.
, « Effects of Tityus serrulatus scorpion toxin gamma on voltage-gated Na+ channels. », Circ Res, vol. 80, nᵒ 3, p. 363-9, 1997.
, « Increase in the proliferative capacity of human myoblasts by using the T antigen under the vimentin promoter control. », Muscle Nerve, vol. 20, nᵒ 4, p. 437-45, 1997.
, « Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels. », FEBS Lett, vol. 412, nᵒ 3, p. 621-4, 1997.
