Publications
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Filtres: Author is Paccalet, Thomas [Enlever les filtres]
The Electroretinogram May Differentiate Schizophrenia From Bipolar Disorder. Biol Psychiatry. 2019.
Polygenic risk scores distinguish patients from non-affected adult relatives and from normal controls in schizophrenia and bipolar disorder multi-affected kindreds. Am J Med Genet B Neuropsychiatr Genet. 2018;177(3):329-336.
Defective compensation over early dysneurogenesis in schizophrenia and mood disorder. Schizophr Res. 2017.
Electroretinographic anomalies in medicated and drug free patients with major depression: Tagging the developmental roots of major psychiatric disorders. Prog Neuropsychopharmacol Biol Psychiatry. 2017;75:10-15.
The interaction of GSK3B and FXR1 genotypes may influence the mania and depression dimensions in mood disorders. J Affect Disord. 2017;213:172-177.
Childhood abuse and neglect may induce deficits in cognitive precursors of psychosis in high-risk children. J Psychiatry Neurosci. 2015;40(5):336-43.
Cognitive structure from childhood to adulthood in kindreds densely affected by schizophrenia and bipolar disorder. Psychiatry Res. 2015;229(1-2):101-8.
Light evoked potentials measured by electroretinogram may tap into the neurodevelopmental roots of schizophrenia. Schizophr Res. 2015;162(1-3):294-5.
Cluster analysis of cognitive deficits may mark heterogeneity in schizophrenia in terms of outcome and response to treatment. Eur Arch Psychiatry Clin Neurosci. 2014;264(4):333-43.
Common childhood determinants of psychiatric and cardiovascular disorders call for common prevention and clinical research. JAMA Pediatr. 2014;168(1):3-4.
Findings and concepts from children at genetic risk that may transform prevention research and practice in schizophrenia and mood disorders. In: From research to practice in child and adolescent mental health. Lanham, Maryland: Rowman & Littlefield; 2014.
Follow-up of a major psychosis linkage site in 13q13-q14 reveals significant association in both case-control and family samples. Biol Psychiatry. 2013;74(6):444-50.
A protective-compensatory model may reconcile the genetic and the developmental findings in schizophrenia. Schizophr Res. 2013;144(1-3):9-15.