Publications
Export 158 results:
Filtres: Author is Mohamed Chahine [Enlever les filtres]
, « A tryptophan residue (W736) in the amino-terminus of the P-segment of domain II is involved in pore formation in Na(v)1.4 voltage-gated sodium channels. », Pflugers Arch, vol. 445, nᵒ 1, p. 18-24, 2002.
, « Ethanol delays and reverses lysophosphatidylcholine-induced calcium overload in neonatal rat heart cells. », Pflugers Arch, vol. 443, nᵒ 1, p. 48-53, 2001.
, « Gating properties of Na(v)1.7 and Na(v)1.8 peripheral nerve sodium channels. », J Neurosci, vol. 21, nᵒ 20, p. 7909-18, 2001.
, « Homozygous SCN5A mutation in long-QT syndrome with functional two-to-one atrioventricular block. », Circ Res, vol. 89, nᵒ 2, p. E16-21, 2001.
, « Implication of the C-terminal region of the alpha-subunit of voltage-gated sodium channels in fast inactivation. », J Membr Biol, vol. 183, nᵒ 2, p. 103-14, 2001.
, « Novel mechanism for Brugada syndrome: defective surface localization of an SCN5A mutant (R1432G). », Circ Res, vol. 88, nᵒ 12, p. E78-83, 2001.
, « Biophysical phenotypes of SCN5A mutations causing long QT and Brugada syndromes. », FEBS Lett, vol. 487, nᵒ 2, p. 224-8, 2000.
, « The distinct HERG missense mutation L564P causes long QT syndrome in one French Canadian family. », Can J Cardiol, vol. 16, nᵒ 3, p. 307-12, 2000.
, « Electrophysiological characterization of SCN5A mutations causing long QT (E1784K) and Brugada (R1512W and R1432G) syndromes. », Cardiovasc Res, vol. 46, nᵒ 1, p. 55-65, 2000.
, « SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells. », FEBS Lett, vol. 467, nᵒ 1, p. 12-6, 2000.
, « Cysteine scanning analysis of the IFM cluster in the inactivation gate of a human heart sodium channel. », Cardiovasc Res, vol. 42, nᵒ 2, p. 521-9, 1999.
, « Modulation of HERG potassium channel properties by external pH. », Pflugers Arch, vol. 438, nᵒ 3, p. 419-22, 1999.
, « Thioridazine lengthens repolarization of cardiac ventricular myocytes by blocking the delayed rectifier potassium current. », J Pharmacol Exp Ther, vol. 288, nᵒ 3, p. 1261-8, 1999.
, « Electrophysiological study of chimeric sodium channels from heart and skeletal muscle. », J Membr Biol, vol. 164, nᵒ 1, p. 25-34, 1998.
, « Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site. », Biophys J, vol. 75, nᵒ 1, p. 236-46, 1998.
, « Effects of Tityus serrulatus scorpion toxin gamma on voltage-gated Na+ channels. », Circ Res, vol. 80, nᵒ 3, p. 363-9, 1997.
, « Increase in the proliferative capacity of human myoblasts by using the T antigen under the vimentin promoter control. », Muscle Nerve, vol. 20, nᵒ 4, p. 437-45, 1997.
, « Myotonic dystrophy kinase modulates skeletal muscle but not cardiac voltage-gated sodium channels. », FEBS Lett, vol. 412, nᵒ 3, p. 621-4, 1997.
, « Restoration of fast inactivation in an inactivation-defective human heart sodium channel by the cysteine modifying reagent benzyl-MTS: analysis of IFM-ICM mutation. », Biochem Biophys Res Commun, vol. 233, nᵒ 3, p. 606-10, 1997.
, « Electrophysiological characteristics of cloned skeletal and cardiac muscle sodium channels. », Am J Physiol, vol. 271, nᵒ 2 Pt 2, p. H498-506, 1996.
, « Okadaic acid enhances prepulse facilitation of cardiac alpha 1-subunit but not endogenous calcium channel currents in Xenopus laevis oocytes. », Can J Physiol Pharmacol, vol. 74, nᵒ 10, p. 1149-56, 1996.
, « Sea anemone toxin (ATX II) modulation of heart and skeletal muscle sodium channel alpha-subunits expressed in tsA201 cells. », J Membr Biol, vol. 152, nᵒ 1, p. 39-48, 1996.
, « Characterizing the mu-conotoxin binding site on voltage-sensitive sodium channels with toxin analogs and channel mutations. », Receptors Channels, vol. 3, nᵒ 3, p. 161-74, 1995.
, « Functional expression and properties of the human skeletal muscle sodium channel. », Pflugers Arch, vol. 427, nᵒ 1-2, p. 136-42, 1994.
, « Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation. », Neuron, vol. 12, nᵒ 2, p. 281-94, 1994.
